VIVA GUIDE TO FIRST PROF (PART-1)- ANATOMY, BIOCHEM, PHYSIOLOGY...
Hey first years! I actually had all the bank of VIVA questions of the last year saved. What we did was that we asked everyone in the class to come online and share his/her experiences and questions on the facebook group of our class after taking the vivas. So I am giving you all that had been uploaded by my class fellows. I just made a doc of each subject and compiled everyone's contribution.
Here it is. Just don't get intimidated by the length of it :P Some questions might repeat. I hope you'll have a good idea of what happened in last year's prof and how :) So at least, don't skip the questions mentioned below.
Best of luck :)
Anatomy guide and VIVA questions for Prof
§ Major viva.Main focus should be on chalking of
bones, joints and movements and nerve supply , actions while preparing for
viva.Be on time , surface marking starts at exact 7:30.Us k baad people are
called into L.R.C in groups , each person is handed a bone.ab sab se pehle you
have to mark the attachments , there is ample time.Phir Maam Attiya takes the
external , she asks three, four points about the bone given to you.Mujhse
capsular attachments karai thi and nerve supply of muscles attached.Then
adjunct , conjunct movements of knee joint.Then aik aadh question embryo ka and
you are good to go , shes such a sweetheart.(Maam attiya has lower limb ,
embryo and g.a)Us k baad you have to wait around in the L.R.C for Maam Raafia
ki turn.Just relax back on the chairs and revise the limb on display apney sath
waley bandey k sath.Maam Raafia calls two bachey aik sath.One has to do the
spotting on the limb on display ( She has upper limb , thorax so upper limb was
on display.)Gandi kaali body thi but muscles identify hogaye they , she points
on the muscles and gives you around a minute to decide what they are while she
takes the viva of the other kid.Utni dair mein you should identify the muscles
and make a mental sketch of their nerve supplies , actions cause when she'll
come around again , yehi poochein gi.e.g agar pronator teres hai tu phir her
next question will be what nerves passes b/w these two heads , associated
syndrome)Then its your turn for viva.Visceras , lung impressions , bucket
handle , pump handle movements , typical atypical ribs , heart k features
general , sternum and ribs ki attachments aani chahiye :)Joints k axises
poochey they , ulna k joints and types , whether thymus is present in adults or
not , bas superficial si cheezain poochein :)Bas us k baad aap rafu chakar.
;)Now half the people do spotting first , then the rest.Keep contact and ask
the pehle group waley to text all the spots , hogayi anatomy.Keep your cool ,
Maam Raafia baar baar pochein gi if " you are sure" , nod and say "yes"
, inshallah sab pass hongein.Spotting questions.Evertors, name them?Artery most
commonly affected in MI?Nerves forming deep cardiac plexus?Nerves injured in
cubital tunnel syndrome?Radiographs pe obturator foramen , pubic symphysis pe
spots they. Oligohydroamnios and causes,
§ questions asked to me by mam attia: attachments on tibia, types of decidua and
definition, growing end of bone, sendups ma marks :P difference between primary
and secondary spermatocyte, difference of types of villi
§ percentage of tubal pregnancies, fate of
neural crest cells, from where adrenal cortex is derived, joints formed by hip
bone, their types, difference between primary and secondary cartilaginous
joints, movements at ankle joint
§ femur atachments (marking) , blood supply of
neck of femur, growing end, y growing end, secondary centers of ossification,
extension of knee kon krwata? rectus femoris is different from other extensors
hw? sacrococcygeal teratoma
§ mam raafia: describe impressions n hilum of left lung,
spotting on limb of palmaris longus and ulnar nerve, blood supply of heart,
perform movements of bucket handle and pump handle (she'll hand u a rib n would
say which rib is it and then ask u to perform the movemnt by that rib. For e.g
6th rib pe pump handle n 7 th pe bucket handle movement ho gi) , courses of
nerves
§ supination and pronation movements, their axis
and from where their axis pass in hand, smith's fracture, extensor expansion,
action of trapezius, axis of pump handle movement, joints involved, range of
movement at manubriosternal joint
§ left lung identification, hold heart in
anatomical position, find left atrium on heart, bronchopulmunary segments,
spotting of deltoid, nerve supply of it, other muscle supplies by this nerve,
action of muscle
§ right lung identification, and a very weird
question "wat structures cover the lungs"... "maam visceral
pleura parietal pleura ... and thoracic cage (still she asked for more) maam
deep fascia superficial fascia and finally skin...attachments on humerus, nerve
supply of bracialis. meaning of "proprioceptive" sarcococcegeal
teratoma, flexors of hip joint( dont forget to mention rectus femoris),
difference between primary and secondary oocyte. Spotting was easy and do
revise the hand models. => first rib etcDO study atlas for lung etc cause
maam zarur ID krwati hain. And well, be confident.
Surface Marking - Flexor Retinaculum
External - Fibula ki side determination and the joints it makes
+ their types, cutanoeus innervation of sole, decidua
Internal - Movements of Scapula, apex and base of heart, 2nd rib
Spotting - whitlow, ulnar claw hand, muscles of locking,
coronary sinus tributaries
§ FOR HISTO PRACTICALS:first of all u'd b given an unknown slide
(LONG slide/major focusing). U have to adjust it in the microscope. identify
and write down the diagnosis k knsi slide ha. then maam samina would sign the
diagnosis as right or wrong. If right start makin the dia, if wrong , keep
guessing!After drawing u can start preparing the viva abt that slide in ur head
or with the roll num near by for e.g if u have epithelium as slide then u ought
to knw definition of epithelium, basal lamina , types etc. maam rafea would
come to every student one by one ask 3 4 questions n ur done.After this is
spoting of slides. 10 slides worth 10 marks. 0.5 for identification, 0.5 for
reason.7 marks are for major slide. 3 viva 4 dia. 3 for ur copy. 10 for the
slides.So priority while preparing should be first slide identification, then
practice diagrams, then do related viva.
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ext: lower end of tibia (capsular attachment and epiphyseal
line), dorsiflexion n planterflexton, y planterflexion has more range,
derivatives of intermediate mesoderm, why lower end of tibia if fractures is
difficult to heal, layer that develops urinary bladder
int: post surface of ulna, anatomical snuff box, insertion of
!st dorsal int, radial artery in hand, klumpkes paralysis, fun of pect major,
movements of shoulder joint, cadiac dominance, tndon of torodo!
surface marking:dorsalis pedis artery
external:how fibula is different frm othr bones,,ossification
centers of fibula,,years of development of ossification centers....!!!
embryo:derivatives of endoderm
internal:marking of upper end of radius,,origin insertion of
supinator,nerve supply,,function
identify ulnar nerve frm limb,identify left ventricle of
heart,where does the apex of left ventricle lie...!!!
Maam Attiya:
Consequencies of fertilization,Difference between mitosis of zygote n general
mitosis,Muscles having double supply
Same questions are being repeated. If u get a hip bone, the
very first question will be the difference between male and female. Wahi crus
penis wala. Joints,types of anastomosis from ga. consequences of
fertilization,difference of mitosis,germ layers derivatives,somites etc from
embryo. actions of muscles, nerve supply from upper limb. Spottting on heart
and lungs, pericardial sinuses from thorax.External: Tibia, medial side k
muscles, nerve supply, action, arrangement of structures at lower end of tibia,
unki nerve supply, action. Allantois, connecting stalk.Internal: Limb
spotting,unhi muscle ki nerve supply and action, heart, left atrium ki blood
supply,significance of flexor carpi radialis, movements at shoulder joint,
·
Biochem
VIVA questions for prof
siir shakeel is asking
really easy stuffessential and non essential amino acidsstandard and non
standard amino acidsallosteric enzymes and coperate bindingAcidosis and
alkalosis, causes
henderson-haselbach
equation, Pka ?anion gap
enzyme
inhibition
biochemical
roles of vitamins
beri
beri
phspholipids
steroids
and sterols mein differencebile salts
Sir
asked me abt GAGs proteoglycans،glycoproteins n bile acid funtions!
external
was tough because he asked stuff not really mentioned in te books.
bbut
i can only tell what he asked me..
structres
of protiens and bonds involved in it
supersecondary
motifs and epitope
domains
enzymes
inhibitors as
External
asked me abt immunoglobulins,their structure,diff between heavy n light chain
structure,bence jones protein,how r they seprated 4m albumin n urine?proteins
classification,keratin occurance n amino acid most abundant n it?,AG
(albumin,Globulin)ratio,heat coagulation test.
Other
question tyndall effect,coenzymes table,suicide inhibihion n allosteric
inhibition graph curve with reason(distinction question),cobalamin
absorption,pernicious anemia.
external
asked me how heam is produced in body tell all the mechanism and enzymes,level
of Na in body who it is regulated in body,diabetes insipidus,classification of
carbohydrates and GAGS,
Starch
n glycogen structural diff starch n cellulose structural diff,invert sugar with
reason,sucrose structure bna k dekhao,what is hysteria,causes ov metabolic
acidosis,chemical buffers n how r they written.
ROLE
OF FOLIC ACID AND COENZYME IN WHICH IT IS PRESENTROLE OF COBALAMINE AND HOW WE
MEASURE IT IN BODY?MYOGLOBIN AND HAEMOGLOBIN STRUCTURAL DIFFERENCEDOES
MYOGLOBIN RELEASE OXYGEN AND AT WHICH CONCENTERATION OF OXYGENGLOBULAR PROTEIN
AND AXIAL PROTEIN.WHICH TYPE IS PRESENT IN BODY?FORMULA TO FIND OUT WHETHER A
PROTIN WILL BE GLOBULAR OR AXIAL?
Km
& its significance
classification
of enzymes & what is hydratase?
Co
factor of carbonic anhydrase & Urease
GAG's
, proteoglycan , glycoprotien
significance
of SO4 groups in GAG's
How
enzyme decrease energy of activation? Mechanism??
Diff.
b/w Synthase & Synthetase
Effect
of substrate conc. on rate of reaction
therapeutic
uses of Enzymes? in myocardial infarction???
Gibbs
Donnan equilibrium & its significance?
nucleoside,
polynucleotide
Kbi
starch aur glycogen khaey hain?? forms??
Minerals,vitamins
n enzymes are main start wale studnts jo batch mai hain un k liajo akhir mai
hon un se sir carbohydrates n protein n myoglibin etc ziyada suna hain
Serum
Calcium level? Clinical presentation of Calcium deficiency
sir
shakel ny bht acha viva liya h unho ny mjh sy vitamins ki classificatin,
co.enzymes, vitamin.A functions, night.blindness, beri.beri, visual.cycle draw
krvaya, oxidative.phosphorylatins k componentsExternal.Names of water soluble
vitamins, vitamn.B12 absorptin, functins, disease.
MAJOR
VIVA QUESTIONS.EXTERNAL: all viva wos centred around proteins. started off wth
zymogens their examples..? how is pepsinogen activated to pepsin?
endopeptidases. examples? exopeptidases examples.? carboxy peptidases? wot are
ligases, example.?electrophorosis se related questions.! GIT se proteolitic
enzymes.
MAJOR
VIVA QUESTIONS. INTERNAL: cytochrome p450, simple proteins, conjugated
proteins( def +examples),lipoproteins defination examples. membrane proteins ki
nature. LDL,VLDL, HDL, why are HDLs named so? chylomicrons, biotin, biocitin nd
the reaction in which it is utilized, same questions with pento thenic acid nd
thiiamine. dry beri beri(detail), wet beri beri,allosteric inhibition, niacin
acts as coenzyme for?,co enzyme defination.? pyruvate dehydrogenase function,
oxido reductases ki sub classifications,def nd 1 exampe each.! :(
Prctcl
viva: milk residue n filtrate kaisay banta hai, acetic acid milk main daalne se
kia cheez change hoti hai, ph controlling mechanisms in bodyMajor viva Internal:
Metabolic alkalosis, Body main Ca ki forms, What is Tetany, Whyit occurs, level
of ca in body, Pyridoxine detailed, tb k patients main pyridoxine kyun kam
hoti hai , vit E ka name, action, Rancidity, name of Fatty acids,External:
carbohydrates classification, which polysacchacaride occurs in humans, where is
it located, where is it metabolized to form energy, fats ka energy production
main role hai ya nae,How glycogen forms, enzyme acting on it, deficiency of
which vitamins causes anemia, how does pyridoxine causes anemia, heme synthesis
internal=
visual cycle why called a cyclic process.....MOST IMP DEFINE BIOCHMSTRY N ITS
SIGNFCANCE,..:p,:P,,ENZYMES OF VISUL CYCLE..asked me ka enzyme ka nam lo i said
carbonic anhydars he then asked its functn ...etc...then wht r ligases n
relation btwenn ligases n lyase...anion gap signfcnce....retinoic acid.n its
signfnce....
internal....FIGLU
test, Shcilling test, wernickes korsakoff syndrome(in what people r community),
selenecysteine, allosteric effect on enzymes and the reason for the sigmoid
curve.
externa.....vit
D(production,activation,metabolic role,deficincy rsults, osteomalacia, plasma
findings...like alkaline phosphate etc)...whn does acid phosphatase appear in
plasma...conjugated proteins...nucleoproteins.
why
tetany develops in metabolic alkalosis even with normal Ca level,Ca k amount in
body,xenobiotics....
extrnal
askd...whch enzymes are increasad in myocardial infarction and their
timing....essential fatty acids kahan use hotay hain..baqi q easy thay..
external
= classification of fatty acids,iodine numbr,2 classes of complex lipids having
ceramide as their base,lecithins,phospholipases & source of phospholipase A
External
: Vit A maintenance of epithelium kesy krata haior Iron ki absorptionMucosal
Block theoryserum Ca++ level
internal:
polysaccharides, starch n cellulose k dfrnc, glucogen n starch me dfrnc,
cardiolipin, glycerol kya hai, sphingosine kya hai, ceramide kya hai, cyclic
amp, function, koi lipid jo k secondary messanger(diacylglycerole) oxidatv
phosphorylation (def) substrate linked phosphoryltion
external:
buffer, how buffers r formed, example, metabolic acidosis and caussof
respiratry acidosis and compenation, alkali reserve.....
Sir
shakeel: CerobrosidesVit B12, absorption, it's function.Pernicious anemia, why
it is called so.Megaloblastic anemia, why it is called so.Physical Findings in
them.Intrinsic factor, why it is not digested.Mylein degeneration k effects.
External:
Synthesis of heme, it's regulatory enzyme.protoporphyrin ka
structure. difference between uroporphyrinogen 1 and 3.Factors effecting
heme synthesis.Vit b12, it's chemical name, it's coenzymes and their
functions.Schiling's test.
External:
Heme breakdown, porphyrias and their types. Mera to itna sa hi viva tha, iske
baad usne farigh kar dia.External: Vitamers, Ascorbic Acid and its function in
immunityInternal: Km and its significance, Basis of classification of proteins
into alpha beta and gamma classes, Steroid and sterol difference, Function of
Cholesterol and its forms in body, Bence Jones Proteins, Paraproteins,
Selenocysteine, Chiral Carbon, D & L isomers, Dextrorotation and
Levorotation, Oxidative Phosphorylation, Complexes, Why is NAD present in ETC
and not NADP? Mutliple Myeloma.Heptproteins ,oncotic pressure,Milk alkali
syndrome,difference btw coagulation and denaturation of proteins;example of
ketoacids,colloid ,types of colloid,protective colloid,chemical test for vit.A
Prof
VIVA questions for Physiology
·
(EXTERNAL's QUESTIONS)
*what are the types of T-cells?
*Role of supressor T-cells?
*what's Vaccination?
*commonly vaccinated diseases?
*function of lungs besides Respiration?
*dead space?
*value of anatomic dead space?
*importance of Anatomic Dead Space?
*Role of Nose?
*alveolar ventilation?
*formula for alveolar ventilation?
*functional residual Capcity n it's value?
(INTERNAL's QUESTIONS)
*mechanism of muscle contraction?
*hypertrophy?
*hyperplasia?
*Rigor Mortis?
*Isotonic contraction example in body?
*draw JVP
*what is Shock?
*what are the stages of shock?
*compensatory mechanism for shock?
pysio viva qs. latent period... physiologic effect of
acclimatization on body..... types of RNA function of microRNA .Refractory
period .Kidney functions.... Functions of all cell organelles ...Mountain
sickness ,..action potential nd oxyhemog curves...
minor me external asked about kymography procedure n effects of
ions on ncg related to prac.. mam samia asked about abo grouping .. it was the
minor prac... she asked erythroblastosis foetalis.. rh genes and the procedure
of prac
Draw EcG, plasma proteins functions, latch mechanism, atrial n
ventricle pressure curves, all cell organelles, mitochondria function, cell
cycle, hyperimea(dun know wats dis), action potential, shock ki stages n
treatmentExternal:Identification of O2-Hb Dissociation curveFactors shifting
it.Graph of cheyne stoke's breathingTypes of LeucocytesTypes of
lymphocytesFuctions of the typesCD4 & CD8 receptorsType of cells paralysed
in AIDSBend's
Internal:Cardiac ArrestCardiac FailureCauses of failureHeart
blockWenckebach PhenomemonSkin in thermoregulationPyrogens
Internal: functions of cell membrane, biphasic potential,
compound action potential graph, physiological characteristics of cardiac muscle,
conduction of impulses, characteristics of action potential, antidromic effect
External: Identify graph (that was Chenyene Stokes breathing), causes of
Cheyene Stokes breathing and mechanism, micturition reflex and nerves involved,
events in hemostasis, dissolving of blood clot, name chemicals administered to
body to dissolve clot.
Acid base compensatory mechanisms, mismatch of blood transfusion
and its immediate effects (on heart and kidney), acclimitization, Graphs (ECG,
JVP, cardiac cycle), skin thermoregulation :( hypoxic hypoxia and its
causes :/ types of mRNA 3:) urine composition, cardiac
output dependencies.......................
Vasodilators like NO, endothelin, etc., Autoregulation of
heart...
and the difference between megaloblastic and iron deficiency
anemia, plus their effects on mean corpuscular volume, heart rate, etc etc,
high output cardiac failure, cardiac shocks- External
·
JazakAllah Khair :)
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